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Numerous studies have shown that cholesterol is a major component of lipid rafts. The purpose of this study was to establish the effects of cholesterol enrichment on the composition and function of lipid rafts in the mature mouse testis. Isolated lipid rafts were prepared from adult mouse testes by sucrose density gradient centrifugation. In the testes of male mice, the level of cholesterol was approximately 100 times higher in lipid rafts than in the non-raft fractions. Consistent with this, cholesterol-enriched lipid rafts were substantially depleted of a number of proteins, including β1-adrenoceptors, β1-integrin, caveolin-1, flotillin-1, signal transduction proteins (Gp43, G-proteins, Rap1) and translational proteins (eIF4E, eEF2). Importantly, the levels of spingomyelin phosphatase-2 and Sgpl1 mRNAs, which are highly localized in lipid rafts, were substantially decreased in the testes of cholesterol-depleted mice. Interestingly, in parallel with the functional impairment of cholesterol-enriched lipid rafts, the expression levels of mRNAs for the transcription factors GATA-4, TCF7L1 and MTF-1 were also decreased in the testes of cholesterol-depleted mice. Our data demonstrate that in the

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MySQL – Row based Replication

I have a question about MySQL replication. I have master and slave where I have a table ordered so that on master every row has its own record. Now, the problem is that now I need to keep the data and update it, but since I am not deleting a single record, all of the records in the table appear in replication as if I had deleted them. I am using master-master with replication type row-based.
Why is this happening?

A:

I am not deleting a single record

In row-based replication, all you do is “replicate all rows”. Since you haven’t actually deleted anything, each row is replicated as if you did.
What you want instead is “replicate x rows”, where x is the number of records that need to be retained.

Low serum adiponectin levels in lung cancer patients.
Adiponectin is an abundant adipocyte-derived serum protein that is associated with insulin resistance and atherosclerosis, which are important factors in lung cancer. This study was conducted to determine whether serum adiponectin levels in lung cancer patients were altered. Serum adiponectin levels were measured in 164 lung cancer patients (85 females, 79 males) and 163 healthy control subjects (85 females, 78 males) using an enzyme-linked immunosorbent assay. Adiponectin levels were significantly lower in patients with lung cancer compared with the healthy control subjects (5.3+/-1.8 vs. 8.5+/-2.8 microg/mL, p

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